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DiamoxSpecifies that part vi i of the act no longer applies to certain drugs and medicinal preparations. 1. Piepgras A, Schmiedek P, Leinsinger G, Haberl RL, Kirsch CM, Einhaupl KM. A simple test to assess cerebrovascular reserve capacity using transcranial Doppler sonography and acetazolamide. Stroke 1990; 21: 13061311 Dahl A, Lindegaard KF, Russell D, et al. A comparison of transcranial Doppler and cerebral blood flow studies to assess cerebral vasoreactivity. Stroke 1992; 23: 1519 Okudaira Y, Bandoh K, Arai H, Sato K. Evaluation of the acetazolamide test: vasoreactivity and cerebral blood volume. Stroke 1995; 26: 12341239 Dahl A, Russell D, Rootwelt K, Nyberg-Hansen R, Kerty E. Cerebral vasoreactivity assessed with transcranial Doppler and regional cerebral blood flow measurements: dose, serum concentration, and time course of the response to acetazolamide. Stroke 1995; 26: 23022306 Schmiedek P, Piepgras A, Leinsinger G, Kirsch CM, Einhaupl K. Improvement of cerebrovascular reserve capacity by EC-IC arterial bypass surgery in patients with ICA occlusion and hemodynamic cerebral ischemia. J Neurosurg 1994; 81: 236244 Kleiser B, Widder B. Course of carotid artery occlusions with impaired cerebrovascular reactivity. Stroke 1992; 23: 171174 Yonas H, Smith HA, Durham SR, Pentheny SL, Johnson DW. Increased stroke risk predicted by compromised cerebral blood flow reactivity. J Neurosurg 1993; 79: 483489 Kuroda S, Kamiyama H, Abe H, Houkin K, Isobe M, Mitsumori K. Acetazolamide test in detecting reduced cerebral perfusion reserve and predicting long-term prognosis in patients with internal carotid artery occlusion. Neurosurgery 1993; 32: 912918 Chimowitz MI, Furlan AJ, Jones SC, et al. Transcranial Doppler assessment of cerebral perfusion reserve in patients with carotid occlusive disease and no evidence of cerebral infarction. Neurology 1993; 43: 353357 Sorteberg W, Lindegaard KF, Rootwelt K, et al. Effect of acetazolamide on cerebral artery blood velocity and regional cerebral blood flow in normal subjects. Acta Neurochir Wien ; 1989; 97: 139145 Lang J. Skull Base and Related Structures: Atlas of Clinical Anatomy. Stuttgart: Schattauer; 1995 12. Huber P, Handa J. Effect of contrast material, hypercapnia, hyperventilation, hypertonic glucose and papaverine on the diameter of the cerebral arteries: angiographic determination in man. Invest Radiol 1967; 2: 1732 Poulin MJ, Robbins PA. Indexes of flow and cross-sectional area of the middle cerebral artery using doppler ultrasound during hypoxia and hypercapnia in humans. Stroke 1996; 27: 2244 Kleiser B, Scholl D, Widder B. Doppler CO2 and Dismox test: decreased reliability by changes of the vessel diameter? Cerebrovasc Dis 1995; 5: 397402 Lindegaard KF, Lundar T, Wiberg J, Sjoberg D, Aaslid R, Nornes H. Variations in middle cerebral artery blood flow investigated with noninvasive transcranial blood velocity measurements. Stroke 1987; 18: 10251030 Newell DW, Aaslid R, Lam A, Mayberg TS, Winn HR. Comparison of flow and velocity during dynamic autoregulation testing in humans. Stroke 1994; 25: 793797 Valdueza JM, Draganski B, Hoffmann O, Dirnagl U, Einhaupl KM. Analysis of CO2 vasomotor reactivity and vessel diameter changes by simultaneous venous and arterial Doppler recordings. Stroke 1999; 30: 8186 Kontos HA. Validity of cerebral arterial blood flow calculations from velocity measurements. Stroke 1989; 20: 13 Giller CA, Bowman G, Dyer H, Mootz L, Krippner W. Cerebral arterial diameters during changes in blood pressure and carbon dioxide during craniotomy. Neurosurgery 1993; 32: 737741 Brooks DJ, Redmond S, Mathias CJ, Bannister R, Symon L. The effect of orthostatic hypotension on cerebral blood flow and middle cerebral artery velocity in autonomic failure, with observations on the action of ephedrine. J Neurol Neurosurg Psychiatry 1989; 52: 962966 Caekebeke JF, Ferrari MD, Zwetsloot CP, Jansen J, Saxena PR. Antimigraine drug sumatriptan increases blood flow velocity. DIAMOX, DIAMOX SEQUELS CATAPRES TENEX ALDOMET LONITEN SERPASIL QUESTRAN COLESTID LESCOL LOPID MEVACOR NIACIN LIPITOR M PA: Subject to tablet splitting. Tried and failed OR contraindications to at least two preferred alternatives. A For 80mg dose, use 2 x 40mg tablets. Subject to tablet splitting Limit: One tablet per day PA: Tried and failed or contraindications to lovastatin. Tablets or patches covered. DALMANE 13 Dapsone 9 DAPSONE 9 DARAPRIM 8 DARVOCET-N 12 DARVON PLAIN, CMPD 12 DECONAMINE SR 21 DELTA-CORTEF 16 Demecarium 19 DEMEROL 12 DEMULEN 19 DEPAKENE 11 DEPAKOTE 11 Desipramine 13 Desogestril Ethinyl Estradiol 19 Desoximetasone 0.25% 14 Desoximetasone cream gel .05% 14 DESQUAM X 13 DESYREL 13 Dexamethasone Neomycin 20 Dexamethasone Poly Neomycin 20 Dexchlorpheniramine 21 DIABENESE 16 DIABETA 16 DIAMOX 11 DIAMOX SEQUELS 11 Diazepam 12, 17 DIBENZYLINE 10 Diclofenac Sodium 17, 20 Dicloxacillin 8 Dicyclomine 16 DIDRONEL 16 Dienestrol 18 DIENESTROL 18 Diethylstibestrol 18 DIFFERIN 13 DIFLUCAN 150mg, limited to 1 tablet per co-pay. P.A. required for all other strengths ; 7, 8 Digoxin 11 Dihydrotachysterol 18 DILACOR XR 10 DILANTIN 11 DILATRATE SR 11 DILAUDID 12 Diltiazem 10 Diltiazem SA 10 Diltiazem SR 10 DIOVAN 10 DIOVAN HCT 10 DIPENTUM 17 Diphenhydramine 21 Diphenoxylate Atropine 16 Dipivefrin 19 DIPROLENE 14 DIPROSONE 14 Dipyridamole 17 DISALCID 17 Disopyramide 9 Disopyramide CR 9. Abstract. Clinical features of 27 cases of Chronic Mountain Sickness CMS ; from the Himalayas are reported. They are compared with 75 native highlanders NH ; . All CMS patients were immigrants to high altitude. Mean duration of stay at high altitude was seven years. Mean values for haematocrit and haemoglobin were 80% and 23 G% respectively for the CMS group and .z $&w7.9 G% respectively for the native highlanders group. Mean QRS axis in the -tks -tatter + 7 .'hfcldenee and quantum of protienuria were significantly higher in the CMS group. Cardiac catheterisation studies done in eight CMS cases showed elevated Pulmonary Artery PA ; pressures even after a mean of 14.2 days at sea level. The disease which has four diagnostic elements-hypoxemia and polycythemia, pulmonary hypertension, right ventricular enlargement and nephropathy with dense proteinuria-is a variant of 'Monge's Disease' and a name CMS Phobrang Type is suggested, along with a new approach to clinical classification which may help in diagnosis before cor pulmonale sets in. Limited therapeutic trials conducted at high altitude seem to indicate that yogic deep breathing exercises, low-dose aspirin and diamox may be beneficial in the prevention and therapy of CMS Phobrang Type at h ~ altitude. Antibiotics Antidiarrheal drugs eg loperamide ; Acetaminophen Tylenol ; or aspirin Anti-inflammatory drugs eg ibuprofen ; Antihistamines for hay fever and allergic reactions ; Antibacterial ointment eg Bactroban ; for cuts and abrasions Steroid cream or cortisone for poison ivy and other allergic rashes ; Bandages, gauze, gauze rolls Adhesive or paper tape Scissors, safety pins, tweezers Thermometer Pocket knife DEET-containing insect repellent for the skin Permethrin-containing insect spray for clothing, tents and bed nets Sun block Oral rehydration salts Iodine tablets for water purification ; Syringes and sterile needles Acetazolamide Damox ; for altitude sickness Bring medications in their original containers, clearly labeled. A signed, dated letter from your physician describing all medical conditions and medications, including generic names is also a good idea. If carrying syringes or needles, be sure to have a physician's letter documenting their medical necessity and dulcolax. Nancy dunne index to site resources directory glossary conventional medical therapies for pcos and infertility polycystic ovarian syndrome and infertility is typically treated with a variety of medications and surgical therapies. Thyroid molecular and cellular biology Oral GHRELIN INHIBITS in vitro THYROID CELL FUNCTION B. Holst1, M.-L. Hartoft-Nielsen2, S.K. Rasmussen2, D. Woldbye3, U. Feldt-Rasmussen2 1 University of Copenhagen, Laboratory of Molecular Pharmacology, Department of Pharmacology, Copenhagen 2 University of Copenhagen, Department of Medical Endocrinology, Rigshospitalet, Copenhagen 3 University of Copenhagen, Laboratory of Neuropsychiatry, Department of Pharmacology, Copenhagen, Denmark Ghrelin is a bioactive peptide from the upper gastrointestinal tract, which is secreted during fasting and especially immediately before a meal. The most recognized action of ghrelin is to increase appetite through stimulation of Agouti related peptide AgRP ; and neuropeptide Y NPY ; neurons in the arcuate nucleus of the hypothalamus. However, ghrelin has a pronounced effect on body weight, which cannot be explained by its effect on food intake. Thus, for example the adipocyte appears to be another important target for ghrelin as it decreases lipolysis either through a direct effect on the adipocytes or via the effect of ghrelin on the autonomous nervous system. Still, the effect of ghrelin on lean body mass cannot be explained through these mechanisms. Since the ghrelin receptor is in fact highly expressed in the thyroid gland we hypothesise that ghrelin could be a physiological co-regulator of thyroid function. Administration of ghrelin 10-7 M ; to monolayer cultured human thyroid cells isolated from para-nodular tissue decreased TSH stimulated thyroglobulin release by 77% 18. Ghrelin treated cells 351 2, 0213, ; g L, median range control cells 3, 455 1, ; g L, n 5 ; Ghrelin also decreased TSH stimulated cAMP release from the primary thyroid cell cultures. Ghrelin treated cells 185 124258 ; nmol L; control cells 213 133293 ; nmol L, n 5 ; . RT-PCR experiments showed that ghrelin also decreased the mRNA expression of thyroglobulin, the TSH receptor, the sodium-iodide symporter and thyroid peroxidase in these cells. Ghrelin had no influence on interleukin-6 expression in the cells. It is concluded that these studies add the thyroid gland as a potential target through which ghrelin may act to preserve energy by a decrease in thyroid function during starvation and ditropan. If you did not take one of your usual medications up until the time of your arrival at hss, we may ask you to take one here, prior to your surgery.
Composite Renal Events Calendar Year of Trial Report 1999 2000 2001 Relative Risk 95% CI ; 2.11 0.78-5.73 ; 1.65 1.47-1.86 ; 1.64 1.46-1.84 ; 1.60 1.29-1.98 ; 1.50 1.23-1.83 ; 1.46 1.24-1.73 ; 1.53 1.33-1.76 ; 0.75 1.0 1.5 P Value .14 .001 and rocaltrol. Presentation by Dr. Carole Sable Background Preclinical Microbiology Clinical Pharmacology Clinical Efficacy Clinical Safety. ABILIFY Accutane * Acebutolol Acetazolamide Acetic Acid HC Otic Acetic Acid Otic Aclovate * ACTIVELLA ACTONEL ACTONEL w CALCIUM ACTONEL WEEKLY ACTOS ACULAR Acyclovir Adalat * ADDERALL XR Adderall * ADVAIR ADVAIR HFA ADVICOR AEROBID-M AGENERASE AGGRENOX AKINETON ALBENZA Albuterol Inhaler Albuterol Nebules Albuterol Tab ALDACTAZIDE 50mg Alesse * ALKERAN Allegra * Allopurinol ALOCRIL ALOMIDE ALPHAGAN P Alprazolam ALTACE ALUPENT MDI Amantadine Amaryl * Ambien * Amcinonide Amiloride Amiloride HCTZ Amino Acid Urea Aminophylline Amiodarone Amitriptyline Amoxicillin Ampicillin ANDRODERM ANTABUSE Anthralin Cream APAP Codeine Arava * ARICEPT ARIMIDEX ARMOUR THYROID ARTHROTEC P M M ASACOL Aspirin Codeine Aspirin 800 CR Aspirin 975 EC ASTELIN Atenolol Atenolol Chlorthal ATRIPLA Atropine Ophth ATROVENT MDI Augmentin * AVALIDE AVANDAMET AVANDARYL AVANDIA AVAPRO AVC AVELOX Aygestin * Azathioprine AZELEX AZMACORT AZOPT AZULFIDINE EC Bacitracin Baclofen Bactrim * BACTROBAN CREAM BACTROBAN NASAL Benazepril Benazepril & HCTZ BENTYL SYRUP BENZACLIN Benzamycin Benzocaine Otic Benzocaine-Antipy-PE Benztropine Betamethasone Betaxolol Bethanechol BETOPTIC-S Biaxin XL * Biaxin * Bicitra * Bisoprolol Bisoprolol HCTZ BLEPHAMIDE OPTH BONIVA 150mg Brontex * Bumetanide Bupropion Bupropion-SR Buspirone Butalbital APAP BYETTA CAFERGOT SUPP CAPITROL Captopril Captopril HCTZ CARAC CARAFATE SUSP Carbachol Ophth Carbamazepine Carbidopa Levodopa Carisoprodol Carisoprodol ASA Carteolol Ophth CASODEX CATAPRES-TTS CEDAX CEENU Cefaclor Cefadroxil Cefpodoxime Tab Cefprozil Ceftin * Celexa * CELLCEPT CENESTIN Cephalexin CERUMENEX CETAPRED Chloral Hydrate Chloramphenicol Ophth Chlordiazepox Clindin Chlordiazepoxide Chloroquine 500mg Chlorothiazide Chlorpromazine Chlorpropamide Chlorthalidone 25mg Chlorthalidone 50mg Chlorzoxazone Cholestyramine Ciclopirox Lotion Cimetidine CIPRO HC CIPRODEX Ciprofloxacin Ciprofloxacin Ophth ; Citalopram CLEOCIN 75mg CAP CLEOCIN PED SOLN CLEOCIN VAG CLIMARA 0.0375mg CLIMARA 0.06mg Climara * Clindamycin Cap Clindamycin Topical Clobetasol Clomipramine Clonazepam Clonidine Clonidine Chlorthal Clorazepate Clotrimazole Troche Clozapine CODEINE SOL TAB CODEINE SOLN Codeine Sulf. Tab. P Prior Authorization M M M COLAZAL Colchicine Colchicine Probenicid Colestid * COLYMYCIN-S COMBIVENT COMBIVIR CONCERTA Control Solution Coreg * CORTEF 5mg CORTIFOAM Cortisone CORTISPORIN OPTH. Cortisporin Otic * CORZIDE COSOPT COUMADIN COZAAR CREON CRIXIVAN Cromolyn Neb Cromolyn Ophth CUPRIMINE Cyclobenzaprine 10mg CYCLOGYL 0.5% Cyclopentolate Cyclophosphamide Cyclosporine CYMBALTA Cyproheptadine CYTADREN CYTOMEL CYTOTEC Danazol Dapsone DDAVP TABS Depakene * DEPAKOTE DEPAKOTE ER DEPO-PROVERA 150m DERMASMOOTH Desipramine Desmopressin Desogen * Desonide Desoximetasone DETROL DETROL LA Dexamethasone Dexamethasone Opth Dexedrine * Dextroamphetamine DIAMOX SEQUEL DIASTAT Diazepam Diclofenac Diclofenac Ophth Dicloxacillin Dicyclomine M M M and actonel. His allows for an increase in ventilation of about 60% Guyton, 1969 ; . This is an immediate response to oxygen debt. Over a period of several weeks the capacity to increase alveolar ventilation may in crease 600-700%. Pressure in pulmonary arteries is increased, forcing blood into portions of the lu ng which are normally not used during sea level breathing. The body produces more red blood cells i n the bone marrow to carry oxygen. This process may take several weeks. Persons who live at high alt itude often have red blood cell counts 50% greater than normal. The body produces more of the enzym e 2, 3-biphosphoglycerate that facilitates the release of oxygen from hemoglobin to the body tissues Tortora, 1993 ; . The acclimatization process is slowed by dehydration, over-exertion, alcohol and o ther depressant drug consumption. Longer term changes may include an increase in the size of the alv eoli, and decrease in the thickness of the alveoli membranes. Both of these changes allow for more g as transfer. TREATMENT FOR AMS The symptoms of mild AMS can be treated with pain medications for he adache. Some physicians recommend the medication Djamox Acetazolamide ; . Both Diamox and headache me dication appear to reduce the severity of symptoms, but do not cure the underlying problem of oxygen debt. Diamox, however, may allow the individual to metabolize more oxygen by breathing faster. This is especially helpful at night when respiratory drive is decreased. Since it takes a while for Diam ox to have an effect, it is advisable to start taking it 24 hours before going to altitude. The reco mmendation of the Himalayan Rescue Association Medical Clinic is 125 mg. twice a day. The standard d ose has been 250 mg., but their research shows no difference with the lower dose Princeton, 1995 ; . Possible side effects include tingling of the lips and finger tips, blurring of vision, and alterati on of taste. These side effects may be reduced with the 125 mg. dose. Side effects subside when the drug is stopped. Diamox is a sulfonamide drug, so people who are allergic to sulfa drugs such as pen icillin should not take Diamox. Diamox has also been known to cause severe allergic reactions to peo ple with no previous history of Diamox or sulfa allergies. A trial course of the drug is usually con ducted before going to a remote location where a severe allergic reaction could prove difficult to t reat. Some recent data suggests that the medication Dexamethasone may have some effect in reducing t he risk of mountain sickness when used in combination with Diamox University of Iowa, 1995 ; . Modera te AMS requires advanced medications or immediate descent to reverse the problem. Descending even a few hundred feet may help and definite improvement will be seen in descents of 1, 000-2, 000 feet. Twe nty-four hours at the lower altitude will result in significant improvements. The person should rema in at lower altitude until symptoms have subsided up to 3 days ; . At this point, the person has beco me acclimatized to that altitude and can begin ascending again. Severe AMS requires immediate descen t to lower altitudes 2, 000 - 4, 000 feet ; . Supplemental oxygen may be helpful in reducing the effect s of altitude sicknesses but does not overcome all the difficulties that may result from the lowered barometric pressure. GAMOW BAG This invention has revolutionized field treatment of high altitude illnesses. The Gamow bag is basically a portable sealed chamber with a pump. The principle of operat ion is identical to the hyperbaric chambers used in deep sea diving. The person is placed inside the bag and it is inflated. Pumping the bag full of air effectively increases the concentration of oxyg en molecules and therefore simulates a descent to lower altitude. In as little as 10 minutes the bag creates an atmosphere that corresponds to that at 3, 000 - 5, 000 feet lower. After 1-2 hours in the bag, the person's body chemistry will have reset to the lower altitude. This lasts for up to 12 hour s outside of the bag which should be enough time to travel to a lower altitude and allow for further acclimatization. The bag and pump weigh about 14 pounds and are now carried on most major high alti tude expeditions. The gamow bag is particularly important where the possibility of immediate descent is not feasible. OTHER ALTITUDE-INDUCED ILLNESS There are two other severe forms of altitude illne ss. Both of these happen less frequently, especially to those who are properly acclimatized. When th ey do occur, it is usually the result of an increase in elevation that is too rapid for the body to adjust properly. For reasons not entirely understood, the lack of oxygen and reduced pressure often results in leakage of fluid through the capillary walls into either the lungs or the brain. Continui ng to higher altitudes without proper acclimatization can lead to potentially serious, even life-thr eatening illnesses. HIGH ALTITUDE PULMONARY EDEMA HAPE ; High altitude pulmonary edema results from fluid buildup in the lungs. The fluid in the lungs interferes with effective oxygen exchange. As th e condition becomes more severe, the level of oxygen in the bloodstream decreases, and this can lead to cyanosis, impaired cerebral function, and death. Symptoms include shortness of breath even at re st, tightness in the chest, marked fatigue, a feeling of impending suffocation at night, weakness, a nd a persistent productive cough bringing up white, watery, or frothy fluid University of Iowa, 199 5. ; . Confusion, and irrational behavior are signs that insufficient oxygen is reaching the brain. On e of the methods for testing for HAPE is to check recovery time after exertion. Recovery time refers to the time after exertion that it takes for heart rate and respiration to return to near normal. A n increase in this time may mean fluid is building up in the lungs. If a case of HAPE is suspected a n immediate descent is a necessary life-saving measure 2, 000 - 4, 000 feet ; . Anyone suffering from H APE must be evacuated to a medical facility for proper follow-up treatment. Early data suggests that nifedipine may have a protective effect against high altitude pulmonary edema University of Iowa, 1995 ; . HIGH ALTITUDE CEREBRAL EDEMA HACE ; High altitude cerebral edema results from the swelling o f brain tissue from fluid leakage. Symptoms can include headache, loss of coordination ataxia ; , wea kness, and decreasing levels of consciousness including, disorientation, loss of memory, hallucinati ons, psychotic behavior, and coma. It generally occurs after a week or more at high altitude. Severe instances can lead to death if not treated quickly. Immediate descent is a necessary life-saving me asure 2, 000 - 4, 000 feet ; . Anyone suffering from HACE must be evacuated to a medical facility for proper follow-up treatment. CONCLUSION The importance of oxygen to the functioning of the human bod y is critical. Thus the effect of decreased partial pressure of oxygen at higher altitudes can be pr onounced. Each individual adapts at a different speed to exposure to altitude and it is hard to know. Diamox sequelsBuying diamox onlineThe sensation elicited by carbonated beverages in the mouth is a pleasurable and sought-after sensation for many people despite the fact that this sensation is irritating or sometimes even painful. Indeed, the sensation associated with carbonation may be a major hedonic component contributing to the large consumption of carbonated drinks Woodroof and Philips, 1981 ; . It has been debated whether the sensation elicited by carbonated water is of mechanical origin due to bursting CO2 bubbles stimulating mechanoreceptors or of chemogenic origin by the formation of carbonic acid which then stimulates polymodal nociceptors in the oral cavity. Several recent lines of evidence support the latter hypothesis. Of particular interest is the phenomenon of the `champagne blues'. Mountaineers, after taking the carbonic anhydrase inhibitor acetazolamide Diamox ; for mountain sickness, reported that carbonated beverages lacked the tingle and beer tasted like `dishwater' Hannsson, 1961; Graber and Kelleher, 1988 ; . Since carbonic anhydrase catalyzes the conversion of CO2 to carbonic acid by the following reaction: CO2 + H2O H2CO3 HCO3 + H + these reports suggest that carbonic acid is necessary for the tingle of carbonated drinks. Additional support comes from a study in which the stinging burning after-sensation of carbonated water persisted long after it had been expectorated Green, 1992 ; . CO2 gas at concentrations of 50% elicits pain when delivered to the eyes Chen et al., 1995 ; or nasal sinus Cain and Murphy, 1980; Anton et al., 1992 ; , and solutions saturated with CO2 elicit pain when infused into the skin Steen et al., 1992 ; . CO2 excites corneal nociceptors Chen et al., 1995, 1997 ; , polymodal nociceptors in skin Steen et al., 1995 ; , and lingual nerve Komai and Bryant, 1993 ; and chorda tympani fibers Kawamura and Adachi, 1967; Komai et al., 1994 ; in a manner that is blocked by carbonic anhydrase inhibitors. Carbonated water also excites nociceptive neurons in trigeminal subnucleus caudalis Carstens et al., 1998 ; . Therefore, current neurophysiological and psychophysical evidence supports the idea that CO2 evokes. Diamox tablets also contain the following inactive ingredients: • sodium starch glycollate • povidone • calcium hydrogen phosphate • maize starch • magnesium stearate diamox does not contain gluten, lactose, sucrose, tartrazine or any other azo dyes. Non-Formulary Drug P Q Any drug for cosmetic purposes Any investigational or experimental drug Any drug for smoking cessation * ACHROMYCIN V ACIPHEX Q * ACLOVATE * ADALAT CC AEROBID AEROBID-M ALBUTEROL HFA * ALDACTAZIDE * ALDACTONE * ALDORIL * ALESSE * ALLEGRA ALLEGRA-D ALTOPREV Q * AMOXIL * ANAPROX &DS ; * ARAVA P * ARISTOCORT & A ATACAND HCT P ATACAND &HCT ; P AVELOX Q AXERT AXID BIAXIN & XL ; BIDIL BONIVA * BREVICON * BUMEX * BUSPAR * CALAN & SR ; * CAPOTEN * CAPOZIDE CARDENE SR * CARDIZEM CD CADUET * CECLOR CECLOR CD CEDAX CEFTIN TABLETS CEFZIL * CELEXA CIALIS Q CIPRO XR CLARINEX CLARITHROMYCIN * CLEOCIN * CLODERM * CORDRAN COZAAR P * CUTIVATE * CYCLESSA CL NC NC Mail N N N Non-Formulary Drug * CYCLOCORT * CYTOTEC * DARVOCET-N * DAYPRO * DECADRON DEMADEX * DEMULEN * DESOGEN * DESOWEN * DIAMOX TABS * DIFLUCAN DILACOR XR * DIPROLENE * DIPROSONE DITROPAN & XL ; * DIURIL DORYX * DURICEF * DYAZIDE DYNABAC DYNACIN DYNACIRC & CR ; * DYNAPEN * E-MYCIN * E.E.S. * ELOCON ENABLEX * ERYC * ERYPED ESTROSTEP FE FACTIVE * FELDENE * FLORONE * FLOXIN FROVA * HALOG & E * HYTONE HYZAAR * IMURAN * INDOCIN SR INSPRA * ISOPTIN SR ITRACONAZOLE * KEFLEX KEFTAB * KENALOG KETEK * KLONOPIN * LASIX LESCOL LEVAQUIN LEVITRA * LEVLEN LEXAPRO 10mg P Q CL 31 Mail N N N Non-Formulary Drug P Q * LIDEX & E * LOCOID * LODINE &XL ; * LOESTRIN &FE ; * LO-OVRAL * LOPID * LOPRESSOR & HCT LORABID * LOTENSIN * LOTENSIN HCT * LOZOL * LUVOX MAXALT Q * MAXZIDE * MEVACOR Q MICARDIS P MICARDIS HCT P * MIRCETTE * MICROZIDE * MINOCIN MOBIC * MODICON * MODURETIC MONODOX * MONOPRIL * MONOPRIL HCT * NALFON NAPRELAN NASALIDE NASAREL NASONEX NEXIUM Q NIRAVAM NIZATIDINE * NORDETTE * NOR-QD * NORINYL NORMIFLO NOROXIN NUTRACORT OMACOR OMEPRAZOLE Q * ORTHO-CEPT * ORTHO-CYCLEN * ORTHO-MICRON * ORTHO-NOVUM 1 35 50 * ORTHO-NOVUM 7 * ORTHO-TRI-CYCLEN * ORUVAIL * OVRAL OVCON PARCOPA PAXIL 10mg & CR 12.5mg * PCE CL 31 Mail N N Y. 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